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What are the chances of recovery from brain hemorrhage? If so, how much cam be expected?

Findings:

MRI BRAIN

Left frontotemporoparietal craniotomy is seen Cystic encephalomalacic gliotic changes are seen in left luntotemporal lobes Hemosidere deposition is seen in left frontotemporal lobes & right distal sylvian fisure & lett occipital lobe cortex appearing hypointense on gradient sequence. Focal hemosiderin deposition is som in central pons. Cystic gliosis is also seen in left occipital lobe. Moderate dilatation in ventricular system is seen, predominantly lateral ventricle Esco dilatation is also seen in left lateral ventricle.

Atrophy & gliosis is seen in left posterior thalamus. Mild gliosis is also seen in tight posterior thalamus. Chronic infarct is seen in left parsagittal temperocccipital lobe. Diffuse thinning of corpus callosum is seen, Gliosis is seen in splenium

Acute/subacute infarcts are seen in bilateral periventricular white matter involving from lobes & right temporal lobe. Small gliotic lesions are seen in bilateral cerebellar hemispheres. Approx. 4mm thicknes

extraxial fluid is seen over left anterior cerebellar convexity. Bilateral basal ganglia are normal.

No midline shift is seen.

Sella and parasellar regions do not reveal any significant abnormality. Basal and sylvian cisterns are clear

Major intracranial vessels show normal flow voids.

Cerebellar tonsils are normal in location.

ISIPRESSION:

ACUTE/SUBACUTE INFARCTS IN BILATERAL FRONTOPARIETAL SUBCORTICAL & PERIVENTRICULAR WHITE MATTER & LEFT TEMPORAL LOBE HYPOXIC ISCHEMIC

EVENT.

HEMOSIDERIN DEPOSITION IN BILATERAL TEMPORAL & LEFT FRONTAL LOBE

CORTEX & SMALL FOCAL HEMOSIDERIN DEPOSITION IN CENTRAL PONS-SEQUELAE

OF TRAUMA MODERATE COMMUNICATING HYDROCEPHALUS WITH EX-VACUO DILATATION OF

LEFT LATERAL VENTRICLE

LEFT PCA TERRITORY CHRONIC INFARCT DIPPUSE THINNING OF CORPUS CALLOSUM

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